fish and seafood
Glutamine supplements have been used for many ailments such as:
increase energy levels
improving memory, focus and concentration
attention deficit hyperactivity disorder (ADHD)
A study on bone marrow transplant patients found that Glutamine allowed for the patients to gain a more vigorous, less angry and fewer fatigue symptoms. Fatigue and Lethargy go hand-in-hand with the Psychological trauma of Parasympathetic Depression simply because the face becomes slouched into a saddened look I know as a Psychological Facial Conformity Disorder.
‘Researchers at Boston Women’s Hospital, Massachusetts, gave healthy volunteers between 40 and 60 grams of glutamine a day for five days. Glutamine was found to be safe, and participants could solve problems better in performance tests. The amino acid supplement appears to work soon after starting it, and have a greater effect over time. The results of both studies were published in the Journal of Parenteral and Enteral Nutrition’.
If you do decide to medicate with L-Glutamine please consult your doctor immediately if you suffer any of these symptoms:
• blood in the urine
• problems swallowing
when I havent been able to afford to buy it so now I make sure I can afford it! THE NUMBER ONE AMINO ACID and iv took them all in L-Form. DLPA comes in at 2nd place & 5HTP in 3rd place. If there is one side effect I could say it increases mania but id rather be manic than depressed any day!
The psychological trauma of the Parasympathetic nerve in Depression as a mental health disorder can leave you feeling like you've been hit by a car due to the Lethargy it causes. Some people think you can will yourself better. With the help of placebos mentioned in my two eBooks, this is certainly true: Buy the eBooks
Although the causes of Psychosocial Depression are not fully understood, biological, social and Psychological factors play there roles in forming this Facial Conformity Disorder due to Psychosocial (public) Peripheral (sublime) reactions that cause stresses to the body language and face.
While Parasympathetic nerve Psychological Depressive Disorders such as Bipolar Depression and Psychological trauma of clinical Depression, they are helped with counseling and medication. However, some choose alternative methods like Dr. Slagle to choose supplements such as L-Glutamine that in my estimation is far safer than some medications that are prescribed.
According to the University of Maryland Medical Center, Glutamine is the most abundant Amino acid in your body and has a number of biological functions. It aids in the removal of toxins and aids Chronic Inflammation of Psychological traumas via brain development. If you experience Psychological mood swings your body may need increases via L-Glutamine as it is easily depleted from the body/brain barrier by high levels of the stress hormone Cortisol.
Clinical Psychological Depression occurs in several forms. These conditions include:
1. Major Depressive Disorder.
2. Seasonal Affective Disorder (SAD).
4. Postpartum Depression.
5. Bipolar Depressive Disorder involved with mood swings.
According to NYU Langone Medical Center, symptoms of Psychological traumas of Depression differ from person to person. Some people suffer just a few symptoms of Psychological Parasympathetic nerve Depression, while some suffer a wide range of the many symptoms that Parasympathetic Depressive Disorder patients suffer. Symptoms include:
1. Feelings of low self-worth in Sympathetic nerve Anxious thoughts.
2. Mood swings into irritability.
3. Fatigue and Lethargy.
4. Lack of interest in activities that they once enjoyed.
5. Suicidal thoughts and Psychosocial gestures.
Researchers believe a number of factors are involved such as genetics, stressful life changes, drug and alcohol addiction, lack of social support from Psychosocial circumstances, medical illnesses and also changes in the neurochemistry of the body/brain barrier. The latter is where body language and facial structure cause body language a Facial Conformity Disorder via the Parasympathetic nerve reaction of Rest and Digest instead of its Peripheral body/brain neurochemistry of Ignorance to Psychosocial circumstances.
After Half a century where the first formulation of the Monoamine hypothesis was formed, there is now strong evidence that implies that long-term Neurochemical changes in the Medulla Oblongata and brain areas and circuits that mediate the complex Psychosocial cognitive-emotional behaviours. These represent the biological underpinnings of the Psychological trauma of Depression/Anxiety Disorders via an imbalance in the Parasympathetic and Sympathetic nerves, along with the Vagus nerve and Glossopharyngeal nerve. With a large number of clinical studies suggesting pathophysiology, it is now associated with the dysfunctions of the Glutamatergic system. Dysfunctions in the mechanisms regulating clearance and metabolism of Glutamate and cytoarchitectural/morphological of Chronic Inflammation are in a number of brain areas. And that is along with the negotiating of the Harmony of the Parasympathetic and Sympathetic nerves of the cognitive and Peripheral Psychosocial sublime emotional behaviours. Additionally, a plethora of data and knowledge gained from animal models have shown that different types of environmental Psychosocial stresses enhance Glutamate release/transmission in limbic/cortical areas and exert powerful Psychosocial autonomic structural effects, inducing a Dendritic remodeling, a reduction of Synaptic connections and possibly volumetric reductions resembling those that are observed in the depressive Disorders of patients. That is because a vast majority of the neurons and Synapses in the Parasympathetic and Sympathetic reaction areas and circuits use Glutamate as a Neurochemical in the body/brain barrier of the Psychosocial Autonomic and Peripheral Nervous System. It would be limiting to maintain that Glutamate is in some way ‘involved’ in Depressive/Anxiety Disorders; rather it should be seen as the Glutamatergic system as a primary mediator of the Psychological trauma of pathology of Chronic Inflammation. Also: a final pathway for the therapeutic action of Antidepressant medication and supplementation.
A paradigm shift from a Monoamine theory of Depression to a body/brain Plasticity, Arousal and Reward of Psychosocial body language hypothesis focused on Glutamate may represent a substantial advancement in the working theory that drives the research for new medicament and Cognitive Behavioral Therapy (CBT) via Sympathetic Disassociation and Parasympathetic Ignorance. And that is instead of Rest and Digest the Psychological trauma of Depressive Disorders and emotional mood swings. This is due to a lack of Fight or Flight from the Sympathetic nerve that causes the imbalance in this Harmonic area of the body/brain barrier. But despite multiple types of medication being available with Monoamine-based mechanisms of action such as L-Glutamine, there still remain a large number of patients that do not achieve a sustained remission from their depressive Disorder symptoms with Psychiatric drug treatment of experimental novel (Neologism) means! The need for more improved pharmacotherapy’s of treatment-resistant Depression means there is a large space for the development of new compounds with the natural novel mechanisms of Neurotransmitter action such as Glutamate transmission and its related pathways in the body/brain barrier of the Medulla Oblongata. This is where CBT is the main route of treatment towards the Psychosocial interaction of Neurochemicals in the Parasympathetic and Sympathetic nerves that happens in the Counselors negotiation. That negotiation causes a lifting of mood swings in the Psychosocial Peripheral actions on body language, Prosody (emotional tones) and a Facial Conformity Disorder that gets naturally lifted away from a Psychologically saddening trauma of a Depressive facial structure.
Monoamine (Amino acid) hypothesis has been experimented on for 50 years into Chronic Inflammation research of neuropsychiatric Disorders in particular Depressive/Anxiety Disorders, as well as production and experimentation of the vast array of therapeutic medications. With regards to Depression, it is theorized that pathology was due to or accompanied by reduced availability of Monoamines, particularly Serotonin and Noradrenaline of the Sympathetic nerve and that Antidepressants exerted their therapeutic action by increasing extracellular availability of Monoamines, particularly at the level of Synapses which control Neurochemical production. Its research was intrinsically tautological, in its main evidence and was based on the mechanism itself of Monoamine oxidase inhibitors and tricyclic Antidepressants, drugs that acutely increase the availability of Monoamines.
In a rhetoric fashioning over the years, a tautology is a logical argument constructed in such a way that generally repeats the same concept or assertion differently. However, it is the using of different phrasing or terminology that is the rhetoric, that the proposition as stated is logically irrefutable. Furthermore, it is the obscuring of the lack of evidence or valid reasoning supporting the stated conclusion. More experimental Neologism of Psychiatric Historical Insignificance.
In the following years and decades, the theories have had several modifications in the attempt to solve its inherent inconsistencies, the main one being the temporal discrepancy between the immediate effects of the drugs on Monoamines available (minutes, hours) and their therapeutic effects over several weeks. The basis of a ‘Glutamate theory’ over the natural supplement L-Glutamine can be traced back to the early part of the 1990’s where early findings showed that N-Methyl-D-Aspartate Receptor (NMDA-R) Antagonists possess an Antidepressant-like action. However more recently it has been theorized again and again and evolved, integrating research results from many different fields that includes intracellular signaling/mechanisms of gene expression, neurotrophic mechanisms, neurogenesis, Synaptic function and Plasticity, remodeling of neuronal cells/circuitry, into what has been referred to as a ‘Neuroplasticity theory’. Coincidentally, a corollary of these theories is that Neuroplasticity can be Biphasic: adaptive and also beneficial, such as by doing physical exercise and also taking Antidepressants. And that is versus maladaptive that has been observed in neuroimaging brain studies of Psychologically traumatized depressed patients or in animal models of traumatic stress and Psychological traumas of mood swing disorders. An additional corollary, as evidenced by a number of theoretical studies, is that maladaptive Plasticity in animal models that can at least partly be reversed by therapeutic treatments (CBT), including Antidepressant medications.
Although most of the Neuroplastic changes have been detected in the neurons and the body/brain circuitry of the predominant Glutamatergic Neurotransmitter system, we refer to a ‘Monoamine theory’, not a ‘Glutamate theory’ in the