of new brain cells and also that thoughtful stress and the trauma of Anxiety is the most important precipitator of the trauma of Depression. He believes that the antidepressants medicament known as Selective Serotonin Reuptake Inhibitors (SSRI's) that are designed to boost Serotonin levels to help to kick off the production of new brain cells, which allows for the alleviation of Psychological trauma of Depression by lifting the mood and alleviating the trauma of Sympathetic Nerves Anxious thoughts.
Norepinephrine and Epinephrine are caffeine gained from nutrients such as tea and coffee and both give Adrenaline and or Noradrenaline. Both work on the Sympathetic nerve and the latter Epinephrine is the Harmony of the Parasympathetic nerve. This two nerves are directly involved with the body/brain barrier from the Vagus nerve that is fed via the gut but also controls emotional vocal and thoughts of emotion in its control via its connection to the Glossopharyngeal nerve. Thoughtful Psychological Sympathetic Anxiety problems in the trauma of Depression are controlled via these four nerves, as it is with the Psychological trauma of voices in the head due to a lack of Tinnitus.
Glutamate releases mood enhancers against Nitrogenous Compounds from the Sympathetic nerve and may work in conjunction with Caffeine that feeds the Noradrenaline and Adrenaline to the Sympathetic and Parasympathetic nerves. GABA in the Vagus nerve may release vitamin B6 to stimulate Tryptophan conversion in the brain/blood barrier. Glutamate may work to Reuptake Serotonin in the blood/brain barrier.
GABA (Gamma-Aminobutyric Acid) is formed in the gut from Glutamate and transfers to the brain via the Vagus nerve. It acts as an Inhibitory effect via Adrenaline. And in the Parasympathetic notions of Psychosocial emotional behaviour where the Peripheral Nervous system (PNS) is affected via low tonal devices of the Psychosocial crowd Buzz stimulation. This is how Psychosocial Prosody (emotional tones of the Peripheral Nervous Systems emotional sensations) affects the state of the trauma of Depression. GABA also aids the Central Nervous System (CNS).
GABA is produced in the guts to form Enzymes. As an Inhibitory Neurotransmitter it achieves this by slowing down the Harmony of the Sympathetic nerve of Fight or Flight which is the alarm bell of the body/brain barrier. An imbalance of GABA vitamin B6 could be the cause of the trauma of Depression because of its stimulus in the manufacture of Serotonin and how it slows down the Sympathetic nerve action that results in overstimulation of the Rest and Digest of the Parasympathetic nerve. This then causes the hypnotic trauma of Depression and traumatic thoughtful Anxiety confusion via a Facial Conformity Disorder. This then causes the Facial Conformity Disorder of sadness or an overpowering of the glum and feeling grumpy state of Theory of Mind and Embodiment. And that is in the mood of emotional Psychological Psychosocial Ignorance and Disassociation where the latter is of the Sympathetic Harmony of these two nerves.
So saying Vitamin B6 can aid the use of SSRI’s as can yogurts which are a major form of GABA. SSNI’s may aid the production of a B6 vitamin effect in the Parasympathetic nerve. And that alleviates the Psychological trauma of Sympathetic nerve Anxious Psychosocial Depression via its Sympathetic nerve Harmony. This is the cause of a thoughtful trauma of Parasympathetic Depression and the Harmony of traumatic thoughtful Anxiousness.
Dietary influences of Serotonin come from some calcium-rich foods into the blood supply. It is said there are no foods that directly increase your body's supply of Serotonin. Some foods and nutrients however increase the levels of Tryptophan - a direct cause of the production of the Neurochemical Serotonin. Tryptophan is an Amino acid that directly produces Serotonin in the blood/brain barrier. The key issue with Tryptophan being converted to Serotonin is via vitamin B6.
SSRI's work by increasing Serotonin in the brain’s cells. After carrying a message Serotonin is usually reabsorbed by nerve cells know as Reuptake. SSRIs work by blocking ("inhibiting") reuptake, meaning more serotonin is available to pass further messages between nearby nerve cells at the Vagus nerve in the Medulla Oblongata. In this way more Serotonin becomes available to pass further messages in the brain.
SSRI's usually come in tablet form. When you start on them it will be at the lowest dosage thought necessary to treat the Psychological trauma of your Parasympathetic Depression symptoms.
SSRI'S are usually taken for two to four weeks before symptoms of Parasympathetic Depression start to be relieved. Side effects may be felt but it is important not to stop taking this medicament. The side effects usually wear off quickly. If you feel no benefits after 4-6 weeks then specialists will consider increasing the dosage. A course of treatment usually lasts for 6 months, although longer courses are sometimes recommended simply because in some people recurrent Psychological trauma of Depression occurs. These people may be placed on SSRI's indefinitely.
Most people will experience a few mild side effects when taking SSRI’s. These can be troublesome at first, but do improve over time.
Common side effects can include:
1. Feeling agitated, shaky or anxious.
2. Feeling or being sick.
4. Blurred vision.
5. Low sex drive.
6. Difficulty achieving orgasm during sex or masturbation.
7. In men, difficulty obtaining or maintaining an erection.
You will need to see a doctor every few weeks or so when you first start taking SSRI’s to have a discussion on how well this medicament is working. You can contact your doctor at any time if you experience any troublesome or persistant side effects from SSRI’s.
Some of the medications that can interact with some SSRIs include:
• non-steroidal anti-inflammatory drugs (NSAIDs)– a common type of painkiller that includes ibuprofen, diclofenac or naproxen
• antiplatelets – a type of medication used to prevent blood clots, such as low-dose aspirin and clopidogrel
• theophylline – a medication used to treat asthma
• clozapine and pimozide – medications used to treat schizophrenia and psychosis
• lithium – a medication used to treat severe depression and bipolar disorder
• triptans – a type of medication, such as naratriptan, sumatriptan and zolmitriptan, used to treat migraines
• other antidepressants – including tricyclic antidepressants (TCAs), monoamine oxidase inhibitors (MAOIs) and serotonin-norepinephrine reuptake inhibitors (SNRIs)
However, this isn't an exhaustive list of all the medications that can interact with SSRIs, and not all of these interactions apply to all types of SSRI.
Protein-rich foods, such as meat and chicken contain high levels of the Neurotransmitter Tryptophans. It also appears in dairy foods, nuts and other fowl. However levels of both Tryptophan and Serotonin drop after you eat a meal that is packed with Protein. So why is that? According to the nutritionist Elizabeth Somer when we eat a high-protein meal we flood the system with both Tryptophan and its competing Amino acids and all are fight for entry into the brain. Now that means only a small amount of the Tryptophan gets through to the brain. And so Serotonin levels do not rise.
But eat a Carbohydrate-rich meal and your body triggers a release of Insulin. This Somer says causes any Amino acids in the blood to be absorbed into the body, but not the brain. That is except Tryptophan. It remains in the bloodstream at high levels following the consumption of a Carbohydrate meal. This means it can freely enter the brain and cause Serotonin levels to rise, she says.